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EA can alleviate both macroscopic and microscopic colonic inflammation, upregulate the expression of claudin-1 and ZO-1 to repair the intestinal barrier, remold the overall structure of the gut microbiota, and inhibit pro-inflammatory factors IFN-γ, TNF-α, and IL-6 through the MyD88-dependent TLR4 signaling pathway, preventing excessive immune response throughout the body (Wang et al., 2020b)

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Genetic determinants of EGFR-driven lung cancer growth and therapeutic response in vivo